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Skeletal muscle can release medium-chain acylcarnitines (MCACs) into the circulation to remove acyl-CoA, which can be detrimental to cellular function, particularly when the supply of energy substrates exceeds the oxidative capacity of the TCA cycle

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8 For example, in NSCLC, COL11A1 CAFs accumulate at tumor margins, deposit dense ECM components (COL1A1, COL3A1), and physically restrict CD8 T-cell infiltration by interacting with tumor DDR1