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In addition, the Asp-derived metabolite N-acetylaspartate (NAA) has in some contexts been proposed to act as a signaling-related molecule that supports the maintenance of M2-like functions, suggesting that Asp/Asn metabolism may simultaneously connect different levels of pro-inflammatory effects and reparative programs (71)

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Small molecule inhibitors of ARG2 have been shown to upregulate the immune activity and almost completely counteract the immunosuppression, suggesting that ARG2 can be used as a target for the immunotherapy of AML (Mussai et al., 2013)

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