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The accelerated tissue repair factor is straightforwardfaster healing means less time spent in painful recovery phases
This is the most commonly cited mechanism and arguably the most plausible
TAMs shape ferroptotic sensitivity by controlling iron transport, redox buffering, glutathione metabolism, lipid peroxidation pathways, and mitochondrial stress signaling
Long-term deficiency could potentially lead to nerve damage
Similarly, genetic predispositions in GIPR, FTO, and MC4R pathways correlate with responsiveness to GLP-1 peptide therapy
1Stable gastric pentadecapeptide BPC 157 and wound healingPubMed / Frontiers in Pharmacology 2018 2Brain-gut axis and pentadecapeptide BPC 157PubMed / Current Pharmaceutical Design 2016
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