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Many cardiovascular risk factors such as diabetes mellitus, hypertension and hyperlipidemia are demonstrated to stimulate the interaction of Cav-1 and eNOS, reduce nitric oxide production resulting in endothelial dysfunction and subsequently the formation of atherosclerotic lesion 41,42,43
GHK-Cu also exerts anti-inflammatory effects by inhibiting the release of pro-inflammatory cytokines (IL-6, TNF-alpha) and by modulating the activity of the nuclear factor NF-kB
Yates, M
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