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Thus, we speculate that T3 can increase the production of non-12-OH FXR-antagonistic BAs in human likely attributed to the same mechanisms in mice, thereby promoting GLP-1 and insulin production by suppressing intestinal FXR signaling
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[1] [2] Traditionally administered via subcutaneous injection, some research has explored transdermal patch delivery systems as an alternative route of administration
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When I met with Sinclair, he told me he is gearing up to publish a paper about how his lab reversed aging in rodents
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