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Elevated [Ca 2+ ] i in rat GCs may be a result of exendin-4-induced PKA activation, because our experiments demonstrated that exendin-4 no longer caused changes in [Ca 2+ ] i when PKA activity was blocked by H-89 (Fig
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To determine if pharmacological manipulation of autophagy effects SIPS, we treated GSH-depleted cells with the autophagic inducer rapamycin and autophagic inhibitors 3-MA and Baf-A1
The gap with Marek Health: High-engagement model intended for power users
63 GaoM.MonianP.QuadriN.RamasamyR.JiangX
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