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Mice with IRP2 overexpression exhibit motor dysfunction along with increased TfR1, enhanced iron deposition, and upregulation of ferroptosis markers in the SN

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found that monocyte-derived macrophages from patients with CD were less susceptible to cell death induced by phorbol-12-myristate-13-acetate and speculated that this could result in the prolonged presence of pro-inflammatory macrophages in the gut and incomplete resolution of inflammation [9]

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