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Given this complex picture, although mitochondrial dysfunction, suggested by a number of studies [60], is a plausible reason for lactate elevations in ME/CFS, the other possibilities should not be discarded
The pharmacological reality of peptide degradation in the GI tract makes meaningful oral absorption extremely unlikely without specialized encapsulation technology (which is still largely experimental for peptides of this size)
In some contexts, curcumin can also enhance methylation of oncogenic promoters (e.g., mTOR) to suppress tumor growth
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