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It is also strongly pro-angiogenic: in rat and endothelial-cell models it promoted new blood-vessel formation and blood-flow recovery by up-regulating the VEGFR2AkteNOS pathway[2]

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Two drug candidates, deferoxamine and imatinib, were identified from a high-content screen as being able to block SARS-CoV-2 infection and infection-associated ferroptosis (Han et al

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