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suggested that GHK-Cu may upregulate VEGF and encourage endothelial migration and tube formation in laboratory models, with copper itself proposed as a required cofactor for several angiogenic enzyme systems in these settings
We chose 0.10% as a conservative, disclosed dose and we're honest about the limits of the evidence
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Accessed 11 August 2020
Taking note of these additional influences helps us get a better understanding of our own limits
Several papers discuss tendon fibroblast models, angiogenesis-related models, vascular signaling, nitric oxide, and endothelial pathways [4] [6] [8] [9]
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