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Ploemen, I
We hypothesize that semaglutide may regulate BW by indirect neuronal input to key appetite-modulating relay stations, such as the PB, via several independent brain nuclei that are accessed directly by semaglutide
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In GF mice and in antibiotic-treated mice, which have a very depleted gut microbiota, enteric neuron number and activation are reduced 72,246,247 , likely via impairments in Toll-like receptor 4 (TLR4) signaling
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