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Studied for its potential role in supporting balanced inflammatory responses, which may contribute to improved healing and overall physical resilience
Conversely, after APAP injection in hepatic specific PGC-1 overexpressing mice, the high expression of primary ROS scavengers is not able to counteract the hepatic oxidative stress induced by APAP, due to the collapse of GSH reservoir caused by the lowered Gcl-c expression and by the massive utilization of GSH for NAPQI detoxification performed by Glutathione S-Transferases
Frontiers in Molecular Biosciences , 9
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