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rather, literature is providing the clue suggesting that it is a good endeavour to pursue in order to achieve an appropriate pharmacological target that could aim at reversing MetS in parallel with diet and exercise before it onsets T2D
I, however, suspect there may be another reason
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The mechanism is indirect: improved energy supports adherence to diet and exercise, and lipotropic agents support liver fat metabolism
Whether the research subject has optimal GH output or not is largely irrelevant
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