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As discussed, the anti-hyperglycaemic effects of DPP-4 inhibition are mediated via enhancing levels of endogenous DPP-4 substrates, since the inhibitors themselves do not possess any inherent glucose-lowering activity
3), in addition to the essentiality of glutathione that must be imported from host tissues via GshT [9], prompted us to study the putative impact of GpoA on the pneumococcal pathogenesis using an intranasal mouse model to mimic a natural pneumococcal infection [37]
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Hopefully in the future [these results] could help guide the use of GLP-1 RA for pediatric patients with MASH, with the indication of obesity and type 2 diabetes who have the greatest risk for progression, Tou told Healio
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Accumulation of misfolded proteins in the ER triggers the unfolded protein response (UPR), in which C/EBP homologous protein (CHOP) is activated, subsequently activating the caspase family and inducing chondrocyte death 12
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