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and licensed by Lilly, orforglipron opens the door to a new generation of incretin-based therapies more accessible, manufacturable, and globally deployable

Treated groups exhibited synaptic clefts with narrow spacing and sharply defined PSDs, along with presynaptic terminals containing clustered SVs adjacent to membranes

Mechanistically, T3 suppresses Cyp8b1 expression, resulting in increased the levels of Farnesoid X receptor (FXR)-antagonistic bile acids, thereby potentiating GLP-1 production and insulin secretion by repressing intestinal FXR signalling

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