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107 In prostate cancer, anti-androgen therapies such as enzalutamide induce the histone reader ZMYND8, which cooperates with FOXM1 to activate neuroendocrine transcriptional programs (e.g., ASCL1), facilitating transdifferentiation into neuroendocrine prostate cancer (NEPC)

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The authors confirmed that AngIV analogs can inhibit HGF binding to c-Met and HGF-dependent signaling by interfering with HGF dimerization, a crucial step for c-Met activation

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