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Increased consumption of fatty acids close to the lactate threshold, resulting in improved body composition

That approach is theoretically sound but creates a non-pulsatile GH profile on the days between ipamorelin injections, which may reduce the amplitude of the somatotroph response to ipamorelin over time due to receptor desensitization [7]

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I believe that the notion of oral GLP-1s being a net negative for Stevanato is pretty misguided, for several reasons

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