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We found that certain pathways, including the homolactic fermentation, PWY-5484 (glycolysis II from fructose 6-phosphate), and THISYN-PWY (super pathway of thiamine diphosphate biosynthesis I), as well as general glycolysis pathways, were significantly more prevalent in the control group compared to the vitiligo group at baseline [Figure 6a]
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[4] Read carefully, this paper cuts both ways
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To systematically evaluate the pathways driving this sensitivity, we performed continuous gene set enrichment analysis (GSEA) ranking all mapped genes by their Pearson correlation coefficient ( R ) between basal expression (log 2 (TPM + 1)) and the PRISM log 2 (fold de-enrichment) across the 470 matched cell lines (note that 5 cell lines were not listed on HPA and were therefore not included
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