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doi:10.1038/nrendo.2012.140 7

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We conducted a wide range of additional control experiments to make sure we were looking at a true Orai1 deficiency phenotype. The investigators also created genetically modified mice that lacked Orai1 channels in excitatory neurons, but retained them elsewhere in the body and the brain

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Sergey Terushkin, MD, FACS Many patients compare themselves to dramatic before-and-after photos online

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Myocardial infarction subtypes in patients with type 2 diabetes mellitus and the effect of Liraglutide therapy (from the LEADER Trial)

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Monitoring temperature changes and discussing any concerns with a healthcare provider ensures that the medications benefits outweigh any potential side effects

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GLP-1 receptor antagonist exendin-(9-39) elevates fasting blood glucose levels in congenital owing to inactivating mutations in the ATP-sensitive K+ channel

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