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Studies in Crat knockout mice demonstrated that CrAT deficiency leads to abnormal fuel selection, which results in a perturbation of glucose homeostasis and suggest that deficits in CrAT activity might contribute to diet-induced metabolic inflexibility by exacerbating the Randle glucose-fatty acid cycle 66

TB-500 retains sequence motifs associated with actin cytoskeleton modulation and cellular migration pathways

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10.1186/s40560-014-0067-y 298 TakeokaM.SakaiA.UedaG.GeR

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