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Of note, exendin-phe1 acted as a competitive antagonist against the -arrestin response to GLP-1 itself, suggesting that it might reduce the in vivo -arrestin recruitment by endogenous GLP-1 (Supplementary Fig

However, further research is needed to fully understand its mechanism of action and potential therapeutic applications in obesity

doi:10.1038/s41598-019-53134-0 162

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There is a well-known set of analyses reporting broad transcriptional shifts in fibroblasts exposed to the complex, sometimes framed through comparison against reference expression databases