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Studied in models involving hepatocyte growth factor pathway activity, c-Met receptor signaling, and neurotrophic response systems
PMID: 24762441
Together, these multidimensional strategies hold promise in dismantling the plastic, drug-tolerant tumor cell states that underlie therapy resistance and disease progression
& Igarashi, M
The rationale for this approach was to investigate if GLP-1 signalling could cause metabolic reprograming that would persist after removal of receptor stimulation (Fig
Neural generators of the auditory brainstem response
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