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Similarly, insulin resistance displayed by the db/db mouse model of T2DM [153] contributed to -synuclein accumulation in the midbrain and substantia nigra [154]
When these unstable molecules accumulate faster than the body's defenses can neutralize them, oxidative stress occurs, which may contribute to cellular damage over time
sympathetic nervous system activation secondary to GLP-1 and glucagon receptor stimulation has been proposed, but this remains uncertain
Renee Klee has been helping me with the medical side of things and she is really amazing
Studies have shown that these medications can reduce the risk of major adverse cardiovascular events, including heart attack, stroke, and cardiovascular death
J Infect Public Health 16:67379
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