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These mechanisms seem to be, at least in part, mediated through the modulation of the GH/IGF-1/insulin system

In contrast, induction of ER stress via central administration of thapsigargin completely abolished the RYGB-mediated improvement in leptin sensitivity, without affecting control animals, highlighting ER stress as a key regulatory factor in the beneficial effects of RYGB on hypothalamic leptin signaling [97]

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Wound healing : Studying connective tissue deposition, angiogenesis, and inflammatory markers in animal models

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The mechanism underlying acetaminophen-induced hepatotoxicity in humans and mice involves mitochondrial damage and nuclear DNA fragmentation