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Thus, BED appears to involve GLP-1 dysregulation without consistent endocrine abnormalities, implying that GLP-1 RA therapy works through supraphysiological receptor activation and modulation of central reward circuitry, rather than by correcting a hormonal deficit [55]
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Feb 15 2015;308(4):C297-307
This review aims to synthesize the available data and current insights into this important issue
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