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have reported that Liraglutide ameliorates mitochondrial dysfunction and prevents neuronal loss in the brain of 5FAD mice (188)

reported that GTPSCS can translocate to the nucleus and interact with p300 to promote histone lactylation, a process that depends on the acetylation of the G2 subunit of GTPSCS [95]

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In addition, semaglutide, Liraglutide, and dulaglutide have indications for reducing cardiovascular risk in patients with T2DM and established atherosclerotic cardiovascular disease [113,114,115,116,117,118]

Khodaverdi S, Mohammadbeigi R, Khaledi M, Mesdaghinia L, Sharifzadeh F, Nasiripour S, et al