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Recently, we found that liver-specific knockout of a TGF-/SMAD adaptor II-spectrin (SPTBN1 LSKO) , blocks NASH and HCC, and II-spectrin siRNA reverses NASH in human NASH 3D cultures ( Sci Transl Med

Ammonia is a toxic byproduct of protein metabolism that accumulates in the body and causes cellular damage

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Recent developments of neuroprotective agents for degenerative retinal disorders

These observations qualified the HepG2 model to study the mechanisms described herein

It plays a role in cellular metabolism