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The inhibition, which shows dose dependency, is exerted via interactions with receptors in the brain that are accessible via leaks in the blood-brain barrier of the circumventricular organs, mainly the postrema area, the subfornical organ and the median eminence [10, 11]
The bacteria residing in the intestine may influence L-cell function through various mechanisms, including the production of metabolites that could affect GLP-1 secretion
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