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The digestive symptoms experienced during GLP-1 therapy result from altered gastrointestinal motility and delayed gastric emptying rather than enzyme deficiency
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It reversed TBI-induced loss of dendritic density and synaptic connections
It follows that control of PER1 expression may potentially be employed to reduce tumor growth
The antihyperglycaemic effect starts after the first dose and is sustained throughout the once-weekly dosing interval, as a result of the prolonged half-life of 4.7 days
The pathogenesis of these lesions can be found under the specific disease syndrome
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