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2.2 Substrate-specific metabolic overload (02 h post-meal) Excess glucose engages canonical insulin signaling [insulin receptor substrate (IRS)phosphoinositide 3-kinase (PI3K)protein kinase B (Akt)] to drive glucose transporter type 4 (GLUT4) translocation in skeletal muscle and adipose tissue
Abstract The activation of pancreatic stellate cells (PSCs) is the key mechanism of pancreatic fibrosis, which can lead to -cell failure
Heavy metals enter the living cells and induce oxidative damage to macromolecules by the excessive generation of reactive oxygen species (ROS)
Gold nanoparticles enhance the ability of radiotherapy to induce immunogenic cell death in glioblastoma
Jan 15 2021;28(5):529-537
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