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PPAR- agonist fenofibrate potentiates antioxidative elements and improves oxidative stress of hepatic cells in streptozotocin-induced diabetic animals

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doi:10.1089/jmf.2011.0005

BPC-157s primary signalling mechanisms are well-characterised: it activates VEGFR2, drives downstream ERK1/2 phosphorylation, and stimulates nitric oxide synthesis via the Akt-eNOS axis producing pro-angiogenic, fibroblast-activating, and extracellular matrix-stabilising effects in experimental models

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