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Acknowledgements We would like to thank Hans-Martin Jck (FAU) and Elisabeth Letellier (Uni.Lu) for constructive feedback and continuous support
The process of cisplatin being taken up by proximal tubule epithelial cells (PTEC) and being converted into a potent nephrotoxin initiates the development of cisplatin-induced nephrotoxicity, including AKI, through a transporter-dependent mechanism at the cellular membrane
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A real evaluation first
Constitution [PMID: 16743256] Laboratory literature summary: On Glutathione
Goal: Support anabolic processes and metabolic function through enhanced IGF-1 activity with extended bioavailability [1]
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