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Similarly to the results obtained here, earlier works have reported oxidative stress-related NGF up-regulation in peripheral tissues such as heart (Caporali et al., 2008) and liver (Valdovinos-Flores and Gonsebatt, 2013), that could lead to the observed increase in plasma NGF protein levels after treatment with BSO and iAs (Figure 1)

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3.5 Regulatory mechanisms of ferroptosis in IBD Indeed, the molecular mechanisms of ferroptosis are diverse, and several transcription factors (e.g., TP53, NFE2L2/NRF2, ATF3, ATF4, YAPTAZTFAP2CSPHIFAEPAS/HIFA BACH TFEBJUNHIC1, and HNF4A) play multiple roles through transcription-dependent or transcription-independent mechanisms play multiple roles in shaping ferroptosis susceptibility (28)

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