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All groups exhibited a reduction in HOMA index over the two-month follow-up, suggesting a general improvement in insulin sensitivity
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Our results provide a possible mechanism in which upregulation of phosphorylated p53 and p16 maintain the cell cycle arrest of keloid fibroblasts, and FOXO4-DRI selectively induces apoptosis of keloid senescent fibroblasts by prompting p53-pS15 nuclear exclusion
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Additionally, there is a controlling role of the pentadecapeptide BPC 157 against the tumor-promoting effects of many growth factors and peptides (Sikiric et al., 2018) as well as counteracting harmful escape from the initial healing effect (i.e., angiogenesis)
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